August 2008 鈥� The 12th annual Research Day held by the Department of Cell & Molecular Physiology will focus on the theme of 鈥淗uman Neurological Disorders: From Autism to Alzheimer鈥檚.鈥�
LINK TO RESEARCH DAY PHOTOS >>
This annual event serves to introduce our research to new students and faculty, and to promote productive scientific interactions and collaborations throughout the Health Science programs within and beyond 大象传媒-Chapel Hill. (Download flyer here [pdf].)
- Online registration [Registration Deadline: Monday, August 11 by 5PM]
- Department members: Guidelines for posters and abstracts
Human Neurological Disorders: From Autism to Alzheimer鈥檚
Schedule of Events:
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| Dr. Joe Piven |
10:30AM 鈥� 11:30 AM: 鈥淚maging the Developing Brain in Autism鈥�
Joe Piven, M.D.
Bioinformatics Building, Auditorium (Room 1131)
Dr. Piven is the Sarah Graham Kenan Professor of Psychiatry, Pediatrics, and Psychology, and Director of the Neurodevelopmental Disorders Research Center at 大象传媒 Chapel Hill. Dr. Piven鈥檚 research is focused on the pathogenesis of autism including neural mechanisms, genetic basis and neuropsychological and behavioral phenotypes. His talk with address how early neurodevelopmental disorders contribute to neurological problems and lead to behavioral abnormalities as seen in children with Autism spectrum disorders. Using state-of-the-art brain imaging methods to gain insight into early developmental changes may be helpful in designing cognitive and/or therapeutic interventions to combat Autism.
11:45 AM 鈥� 1:00PM: Lunch for registered attendees
MBRB, Ground Floor Lobby
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| Dr. Sam Sisodia |
1:00 PM 鈥� 2:00 PM: 鈥淔unction and Dysfunction of Presenilins in Alzheimer鈥檚 Disease鈥�
Medical Biomolecular Research Building (MBRB), Ground Floor Auditorium (Room G202)
Dr. Sisodia is the Thomas A. Reynolds Sr. Family Professor of Neurosciences at the University of Chicago. He is a leading expert on the molecular and cell biology of Alzheimer鈥檚 disease pathology. He has been at the forefront of learning how the familial Alzheimer鈥檚 disease (FAD) genes, including the amyloid precursor protein and the presenilins, function normally, and how they contribute to Alzheimer鈥檚 disease pathogenesis. Recently, Dr. Sisodia鈥檚 studies have shown that in mice, exercise has a remarkable ability to protect against Alzheimer鈥檚 disease pathology by favorably changing gene activity in the brain.
02:15 鈥� 03:15 PM: Poster session #1 (2nd Floor Lobby MBRB)
Department members present their research
Guidelines and instructions for posters and submission of abstracts >>
03:15 鈥� 03:45 PM: Coffee break
03:45 鈥� 04:45 PM: Poster session #2 (2nd Floor Lobby MBRB)
Department members present their research
05:00 鈥� 05:15 PM: Poster prize distribution
05:30 鈥� 07:00 PM: Reception at the Carolina Inn for registered attendees
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